>>17643274DHT sensitivity is a term you evidently do not understand.
Nerve cells have an electrochemical resting membrane state. Ions and cations modulate the resting state, through channels that absorb or expunge ions. A nerve cell has to reach a certain voltage in order to be excited enough to initiate its function. That is called the threshold potential. Once the neuron's voltage reaches the state of excitation, it achieves its action potential and performs its function, be it protein production, release of neurotransmitters, or cutting up proteins into enzymes, et cetera.
Repeated action potentials in a neural pathway/network leave the region in Long-term-potentiation. It's easier to achieve action potentials faster, because the neurons are repeatedly excited without having to achieve resting membrane state. That can permeate to satellite neurons as well, thanks to neural facilitation/paired pulse facilitation.
At some point, the neurons undergo long-term-depression, following their sensitization, requiring stronger stimuli to achieve threshold potentials. Unable to achieve that, which is often practically impossible in the first place, leads the neurons to a)be repurposed, b)go dormant, c)undergo apoptosis.
In the case of the nerves in the follicles, they go dormant due to sensitization from too much dihydrotestosterone in the scalp region produced by 5a-reductase.
The same amount of dihydrotestosterone could be promoting hair growth on the face, or pubis, or back, but it leads the neurons on the scalp to go dormant.
>Why does this paradox exist?For the billionth time, because of KRT37, which is absent from the scalp, but present everywhere else. The gene that's expressed by androgens, and its several alleles that are primarily phylogenetically marked, is the primary culprit.
